@prefix this: <http://www.tkuhn.ch/bel2nanopub/RA7Yp5ef-kJ00sjXvKJUcuyXvm5tT1ePk5J7RHAjyFXMM> .
@prefix sub: <http://www.tkuhn.ch/bel2nanopub/RA7Yp5ef-kJ00sjXvKJUcuyXvm5tT1ePk5J7RHAjyFXMM#> .
@prefix beldoc: <http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix rdf: <http://www.w3.org/1999/02/22-rdf-syntax-ns#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix dct: <http://purl.org/dc/terms/> .
@prefix dce: <http://purl.org/dc/elements/1.1/> .
@prefix pav: <http://purl.org/pav/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix belv: <http://www.selventa.com/vocabulary/> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix chebi: <http://www.ebi.ac.uk/chebi/searchId.do?chebiId=> .
@prefix sdis: <http://resource.belframework.org/belframework/20131211/namespace/selventa-legacy-diseases/> .
@prefix obo: <http://purl.obolibrary.org/obo/> .
@prefix occursIn: <http://purl.obolibrary.org/obo/BFO_0000066> .
@prefix species: <http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=> .
@prefix do: <http://disease-ontology.org/term/DOID:> .
@prefix pubmed: <http://www.ncbi.nlm.nih.gov/pubmed/> .
@prefix orcid: <http://orcid.org/> .

sub:Head {
  this: np:hasAssertion sub:assertion;
    np:hasProvenance sub:provenance;
    np:hasPublicationInfo sub:pubinfo;
    a np:Nanopublication .
}

sub:assertion {
  sub:_1 occursIn: do:1936, obo:CLO_0009465, obo:CL_0000115, obo:UBERON_0004535, species:9606;
    rdf:object sdis:monocyte%20adherence;
    rdf:predicate belv:decreases;
    rdf:subject chebi:7583;
    a rdf:Statement .
  
  sub:assertion rdfs:label "a(CHEBI:\"nitric oxide\") -| path(SDIS:\"monocyte adherence\")" .
}

sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements.";
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
    dce:title "BEL Framework Large Corpus Document";
    pav:authoredBy sub:_3;
    pav:version "20131211" .
  
  sub:_2 prov:value "The interaction between monocytes and vascular endothelial cells is enhanced by endothelial cell senescence,33 also promoting atherogenesis. This change appears to be mediated by upregulation of adhesion molecules and proinflammatory cytokines, as well as through decreased production of NO by senescent endothelial cells.18,19,34,35";
    prov:wasQuotedFrom pubmed:17204661 .
  
  sub:_3 rdfs:label "Selventa" .
  
  sub:assertion prov:hadPrimarySource pubmed:17204661;
    prov:wasDerivedFrom beldoc:, sub:_2 .
}

sub:pubinfo {
  this: dct:created "2014-07-03T14:33:07.896+02:00"^^xsd:dateTime;
    pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}