@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix chebi: . @prefix go: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: obo:CLO_0003684, species:9606; rdf:object go:0034976; rdf:predicate belv:increases; rdf:subject chebi:9516; a rdf:Statement . sub:assertion rdfs:label "a(CHEBI:thapsigargin) -> bp(GOBP:\"response to endoplasmic reticulum stress\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "20131211" . sub:_2 prov:value "Lower levels of eIF2alpha phosphorylation in Nck-overexpressing cells were still observed for up to 24 h of treatment with Tn (data not shown) or with the calcium ATPase inhibitor thapsigargin, which also induces ER stress (Tg) (Fig. 1, B and D), demonstrating that Nck overexpression causes a long-lasting inhibition of eIF2alpha Ser51 phosphorylation. "; prov:wasQuotedFrom pubmed:16835242 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:16835242; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:01.747+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }