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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
http://www.tkuhn.ch/bel2nanopub/RA6dldgiV9AJNnKa89gAWeWfR0cPiHzpooCCuk-GzTB_o#assertion
http://resource.belframework.org/belframework/20131211/namespace/selventa-named-complexes/p85%2Fp110%20PI3Kinase%20Complex
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0043234
http://www.tkuhn.ch/bel2nanopub/RA6dldgiV9AJNnKa89gAWeWfR0cPiHzpooCCuk-GzTB_o#_1
http://semanticscience.org/resource/SIO_000139
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http://amigo.geneontology.org/amigo/term/GO:0016301
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http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://www.w3.org/2000/01/rdf-schema#label
kin(complex(SCOMP:"p85/p110 PI3Kinase Complex")) -> p(EGID:5243)
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
http://purl.org/dc/elements/1.1/description
Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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http://www.w3.org/ns/prov#value
Although inhibition of MEK/MAPK or PI-3K/Akt can each enhance the cytotoxicity of paclitaxel, doxorubicin, or 5-fluorouracil, inhibition of the PI-3K/Akt pathway seems to have a greater effect than inhibition of the MEK/MAPK pathway in reversing Ras-mediated drug resistance.
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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2014-07-03T14:32:04.058+02:00
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