[The prediction that K ATP channel overactivity should cause a diabetic state due to undersecretion of insulin has been dramatically borne out by recent genetic studies implicating activating mutations in the Kir6.2 subunit of K ATP channel as causal in human diabetes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
Gene-disease associations inferred from text-mining the literature.
DisGeNET evidence - LITERATURE