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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RA5wZw31B-9Coz5k8uuNCgHG9ZLNd7cluRxiyDPk4uk04#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RA5wZw31B-9Coz5k8uuNCgHG9ZLNd7cluRxiyDPk4uk04#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0042789
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=5010
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://www.selventa.com/vocabulary/translocationTo
http://amigo.geneontology.org/amigo/term/GO:0009986
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=6091
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http://purl.obolibrary.org/obo/BFO_0000066
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tscript(p(HGNC:HMGA1)) -> surf(p(HGNC:INSR))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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Lack of the architectural factor HMGA1 causes insulin resistance and diabetes in humans and mice Restoration of HMGA1 protein expression in subjects' cells enhanced INSR gene transcription, and restored cell-surface insulin receptor protein expression and insulin-binding capacity.
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Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.tkuhn.ch/bel2nanopub/RA5wZw31B-9Coz5k8uuNCgHG9ZLNd7cluRxiyDPk4uk04
http://purl.org/dc/terms/created
2014-07-03T14:30:35.805+02:00
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