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http://www.nanopub.org/nschema#hasPublicationInfo
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RA5jkcdakARorhpnTHxEtDy10rbnDmgW7IssZ1eiDjO38#assertion
http://www.tkuhn.ch/bel2nanopub/RA5jkcdakARorhpnTHxEtDy10rbnDmgW7IssZ1eiDjO38#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.informatics.jax.org/marker/MGI:1334419
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_33697
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http://purl.obolibrary.org/obo/BFO_0000066
http://purl.bioontology.org/ontology/MSH/D008264
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http://purl.obolibrary.org/obo/BFO_0000066
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http://resource.belframework.org/belframework/1.0/namespace/selventa-legacy-chemical-names/Oxidized%20Low%20Density%20Lipoprotein
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http://www.w3.org/2000/01/rdf-schema#label
a(SCHEM:"Oxidized Low Density Lipoprotein") -> r(MGI:Cd5l)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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The regulation of AIM via LXR/RXR suggested that incorporation of oxLDL that yields oxysterols, the native ligands of LXR, may also upregulate AIM expression in macrophages. As expected, incubation of macrophages with oxLDL, but not native LDL, resulted in a significant increase of AIM (cd5l) mRNA.
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RA5jkcdakARorhpnTHxEtDy10rbnDmgW7IssZ1eiDjO38#pubinfo
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http://purl.org/dc/terms/created
2014-07-03T14:30:15.356+02:00
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http://purl.org/pav/createdBy
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