@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix mesh: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:97005; a Protein: . sub:_2 geneProductOf: mgi:891971; a Protein: . sub:_3 occursIn: mesh:D008168, species:10090; rdf:object sub:_2; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(MGI:Mmp12) -| p(MGI:Serpina1a)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "1.4" . sub:_4 prov:value "In response to cigarette smoke, a1AT protein levels were at least as prominent in serum following cigarette smoke exposure in both wild-type and MMP-12-deficient mice (Figure 5, bottom). In the lung, however, a1AT levels were elevated in MMP-12-/- mice, but reduced by 50% in wild-type mice, following exposure to cigarettes (Figure 5, top). "; prov:wasQuotedFrom pubmed:14633606 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:14633606; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:18.486+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }