@prefix dcterms: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix chebi: .
@prefix RNA: .
@prefix hgnc: .
@prefix geneProductOf: .
@prefix species: .
@prefix occursIn: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 geneProductOf: hgnc:2674;
a RNA: .
sub:_2 occursIn: species:9606;
rdf:object sub:_1;
rdf:predicate belv:increases;
rdf:subject chebi:46024;
a rdf:Statement .
sub:assertion rdfs:label "a(CHEBI:\"trichostatin A\") -> r(HGNC:DAPK1)" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_4;
pav:version "1.4" .
sub:_3 prov:value "(from text) Interestingly, treatment of MM cell lines with TSA alone also resulted in up-regulation of important cancer-related genes including ING1p33, TIMP3, CDKN2D, PTENP1, CDKN1C/p57KIP2, DAPK, and WIG1, suggesting that aberrant histone deacetylation is an important mechanism for inactivation of cancer-related genes in MM.";
prov:wasQuotedFrom pubmed:18172295 .
sub:_4 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:18172295;
prov:wasDerivedFrom beldoc:, sub:_3 .
}
sub:pubinfo {
this: dcterms:created "2014-07-03T14:30:52.445+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}