@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix mesh: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:102709; a Protein: . sub:_2 occursIn: species:10090; rdf:object mesh:D009765; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(MGI:Cav1) -> bp(MESHPP:Obesity)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "1.4" . sub:_3 prov:value "A more thorough analysis of Cav-1 null mice revealed that, whereas a caveolin-1 deficiency did not lead to an overt diabetic phenotype, it did result in abnormalities in lipid homeostasis, characterized by resistance to diet induced obesity, ablation of the hypodermal fat layer, progressive white adipose tissue atrophy, and hypertrophy of brown adipose tissue (58)."; prov:wasQuotedFrom pubmed:14607781 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:14607781; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:18.267+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }