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[Our data suggest that the functional complementation of APCL by APC constitutes a substantial facet of tumour development, because the truncating mutations of APC in colorectal tumours from familial adenomatous polyposis (FAP) patients are almost always selected for the retention of at least one 15R.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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