@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix sdis: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix do: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: do:1936, obo:CL_0000115, obo:UBERON_0001969, species:9606; rdf:object sdis:Atherogenesis; rdf:predicate belv:increases; rdf:subject sdis:mitochondrial%20DNA%20damage; a rdf:Statement . sub:assertion rdfs:label "path(SDIS:\"mitochondrial DNA damage\") -> path(SDIS:Atherogenesis)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "20131211" . sub:_2 prov:value "MtDNA damage, in turn, leads to increased ROS production and atherogenesis. 20 For example, mutations in complex I genes lead to increased mitochondrial ROS production.21,22"; prov:wasQuotedFrom pubmed:17332437 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:17332437; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:08.855+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }