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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RA0mA6NIli-V492MCg_dh_G6wGOFtvDUkRdJBJYIgZXsY#_6 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RA0mA6NIli-V492MCg_dh_G6wGOFtvDUkRdJBJYIgZXsY#_5 http://www.w3.org/ns/prov#value In lean mice and db/db mice, injection of insulin produced rapid tyrosine phosphorylation of insulin receptor substrate 1 (IRS-1), recruitment of p85, the regulatory subunit of phosphatidylinositol 3-kinase and phosphorylation of the downstream signaling molecule Akt. CNTF (0.1 mg/kg per day or 0.3 mg/kg per day) treatment increased hepatic insulin sensitivity as evidenced by the activation of AKT, a downstream molecule of PI3K. 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