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http://www.tkuhn.ch/bel2nanopub/RA-bxfUwWcedsZdbrir70OXvUOZlkryxRdZ11cT_T_EP4
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http://www.tkuhn.ch/bel2nanopub/RA-bxfUwWcedsZdbrir70OXvUOZlkryxRdZ11cT_T_EP4#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RA-bxfUwWcedsZdbrir70OXvUOZlkryxRdZ11cT_T_EP4#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0042789
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http://purl.obolibrary.org/obo/RO_0002204
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http://purl.obolibrary.org/obo/BFO_0000066
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http://www.w3.org/2000/01/rdf-schema#label
tscript(p(HGNC:SPI1)) -| p(HGNC:MAFB)
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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BEL Framework Large Corpus Document
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DCs inducing levels of PU.1 inhibited MafB expression in monocytes, which appeared to be required for DC specification, since constitutive MafB expression inhibited DC differentiation. Consistent with this, PU.1 directly bound to MafB, inhibited its transcriptional activity in macrophages, and repressed its ability to induce macrophage differentiation
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Selventa
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2014-07-03T14:32:36.511+02:00
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