@prefix this: <http://rdf.disgenet.org/nanopublications.trig#NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix sio: <http://semanticscience.org/resource/> .
@prefix ncit: <http://ncicb.nci.nih.gov/xml/owl/EVS/Thesaurus.owl#> .
@prefix lld: <http://linkedlifedata.com/resource/umls/id/> .
@prefix miriam-gene: <http://identifiers.org/ncbigene/> .
@prefix miriam-pubmed: <http://identifiers.org/pubmed/> .
@prefix eco: <http://purl.obolibrary.org/obo/eco.owl#> .
@prefix wi: <http://purl.org/ontology/wi/core#> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix pav: <http://purl.org/pav/2.0/> .
@prefix prv: <http://purl.org/net/provenance/ns#> .
@prefix dcterms: <http://purl.org/dc/terms/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix dgn-np: <http://rdf.disgenet.org/nanopublications.trig#> .
@prefix dgn-gda: <http://rdf.disgenet.org/gene-disease-association.ttl#> .
@prefix dgn-void: <http://rdf.disgenet.org/v2.1.0/void.ttl#> .
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  this: np:hasAssertion dgn-np:NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q130_assertion ;
    np:hasProvenance dgn-np:NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q130_provenance ;
    np:hasPublicationInfo dgn-np:NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q130_publicationInfo ;
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  dgn-np:NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q130_provenance a np:Provenance .
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}
dgn-np:NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q130_assertion {
  miriam-gene:7015 a ncit:C16612 .
  lld:C0023473 a ncit:C7057 .
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    a sio:SIO_001121 .
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dgn-np:NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q130_provenance {
  dgn-np:NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q130_assertion dcterms:description "[We used two imatinib resistant cell lines, K562-ADM cells, which over-express P-glycoprotein (a product of the ABCB1 gene, more commonly known as MDR1), and K562-hTERT cells, which over-express the telomerase reverse transcriptase (TERT), as models to show that the acquisition of multidrug resistance in CML is associated with the enhanced phosphorylation of signal transducer and activator of transcription 5 (STAT5).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
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    rdfs:comment "Gene-disease associations inferred from text-mining the literature."@en ;
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}
dgn-np:NP873214.RA-ELRmU3VJbqIMxDUruW6-lGwjnWfr_JWj92uwMhqN_Q130_publicationInfo {
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