@prefix dcterms: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix schem: .
@prefix Protein: .
@prefix hgnc: .
@prefix geneProductOf: .
@prefix hasPart: .
@prefix ProteinComplex: .
@prefix species: .
@prefix occursIn: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 hasPart: sub:_2, sub:_3, sub:_4;
a ProteinComplex: .
sub:_2 geneProductOf: hgnc:12744;
a Protein: .
sub:_3 geneProductOf: hgnc:7553;
a Protein: .
sub:_4 geneProductOf: hgnc:6913;
a Protein: .
sub:_5 occursIn: species:9606;
rdf:object sub:_1;
rdf:predicate belv:increases;
rdf:subject schem:D-Glucose%2C%20anhydrous;
a rdf:Statement .
sub:assertion rdfs:label "a(SCHEM:\"D-Glucose, anhydrous\") -> complex(p(HGNC:MLXIPL),p(HGNC:MYC),p(HGNC:MAX))" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_7;
pav:version "1.4" .
sub:_6 prov:value "Glucose increased c-Myc abundance and association with its heterodimer partner Max. Manipulations that prevented the formation of a functional c-Myc/Max heterodimer reduced the expression of the L-PK gene. In addition, glucose augmented the binding of ChREBP, c-Myc, and Max to the promoter of the L-PK gene in situ. #[JL] Chrebp is MLXIPL, and is implicated as a glucose-responsive factor. This is basically saying that glucose activates MLXIPL- activation of MYC";
prov:wasQuotedFrom pubmed:17341548 .
sub:_7 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:17341548;
prov:wasDerivedFrom beldoc:, sub:_6 .
}
sub:pubinfo {
this: dcterms:created "2014-07-03T14:30:49.471+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}