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[Exogenous administration of the satiety factor cholecystokinin (CCK; 8-30 �g/kg, i.p.) dose-dependently reduced food intake in Non-Tg controls and increased inactive behaviour, but had no effect on food intake or behaviour in 3�TgAD mice.CCK (15 �g/kg, i.p.) increased c-Fos protein expression in the supraoptic nucleus of the hypothalamus, and the nucleus tractus solitarius (NTS) and area postrema of the brainstem to the same extent in Non-Tg and 3�TgAD mice, but less c-Fos positive cells were detected in the paraventricular hypothalamic nucleus of CCK-treated 3�TgAD compared to Non-Tg mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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