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[Using a small molecule Nox4/Nox1 inhibitor, we demonstrate that targeting Nox4 results in attenuation of an established fibrotic response, with reductions in gene transcripts for the extracellular matrix components collagen 1?1, collagen 3?1, and fibronectin and in principle pathway components associated with pulmonary fibrosis and hypoxia-mediated vascular remodeling: transforming growth factor (TGF)-?1, plasminogen activator inhibitor-1, hypoxia-inducible factor, and Nox4.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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