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[Although non-tumor-cell-autonomous mechanisms can prevent full eradication of dasatinib-refractory ALL in this clinically relevant model, the emergence of resistance to BCR-ABL kinase inhibitors can be effectively circumvented by the addition of 'conventional' chemotherapeutic agents with alternate antileukemic mechanisms of action.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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