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[Together, these data suggest that a strong selective growth advantage for loss of PTEN and upregulation of PI3K/AKT, combined with the close proximity of PTEN to a large unstable segment of repeated DNA comprising SD17 and SD18, can lead to recurrent microdeletions of the PTEN gene in prostate cancer.� 2011 Wiley Periodicals, Inc.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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